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Archive for 8 mai 2012

Actualités de l’audio semaine 21

Written by genyseb2 on . Posted in Audiologie, DISTRIBUTEURS, Revue de presse

Un petit topo de ce qui se dit sur le web audio !

Une pincée de politique pour commencer…

Pour commencer : le dépistage néonatal qui passe en force par arrêté ministérielle. Suite aux nombreux rebondissants, traduisant une crispation des politiques sur l’article 25 de HPST, le dépistage précoce des troubles de l’audition est adopté. Et vous, qu’en pensez vous ?

%POLL_21%

Autre actualité, lié à une décision politique, la classe PONTO (implant ostéo-intégré d’oreille moyenne) de la société oticon médical a reçu un avis favorable de la HAS ; agence qui statue sur la validité et le service médical rendu des dispositifs médicaux.

Un soupçon d’éco…

Quand AMPLIFON rit (+3,9 % au premier trimestre), AUDIKA pleure (-5,6 % au premier trimestre)… L’exposition uniquement franco-italienne du champion boursier français de l’audioprothése nuit à son chiffre d’affaire qui se replit à 25 Millions d’Euros. Le contexte politique, économique et climatique du premier trimestre n’a pas jouer en la faveur d’AUDIKA.

… et surtout quelques petits infos technicoscientifiques glanées sur le net !

L’exposition répétée à des niveaux de l’odre de 70 dB SPL peut provoquer à long terme une réduction de l’activité neuronale du système nerveux auditif. Cette altération provoque une réduction de l’intelligibilité, sans provoquer de lésions aux niveaux périphériques (audiométrie, PE du tronc cérébral et OEAP normaux !). Cette modification de l’activité du cortex auditif provoque une réorganisation de la carte tonotopique (augmentation de l’activité en dehors des zones fréquentielles sur exposées !). Bref, une personne se plaignant de moins bien entendre avec un audiogramme plat entre 0 et 20 dB peut tout de même souffrir de problème d’intelligibilité (à quand des tests plus sensibles ? ; cet article me fait penser au syndrome de king koperski).

It has become increasingly clear that even occasional exposure to loud sounds in occupational or recreational settings can cause irreversible damage to the hair cells of the cochlea and the auditory nerve fibers, even if the resulting partial loss of hearing sensitivity, usually accompanied by tinnitus, disappears within hours or days of the exposure. Such exposure may explain at least some cases of poor speech intelligibility in noise in the face of a normal or near-normal audiogram. Recent findings from our laboratory suggest that long-term changes to auditory brain function—potentially leading to problems with speech intelligibility—can be effected by persistent, passive exposure to more moderate levels of noise (in the 70 dB SPL range) in the apparent absence of damage to the auditory periphery (as reflected in normal distortion product otoacoustic emissions and auditory brainstem responses). Specifically, passive exposure of adult cats to moderate levels of band-pass-filtered noise, or to band-limited ensembles of dense, random tone pips, can lead to a profound decrease of neural activity in the auditory cortex roughly in the exposure frequency range, and to an increase of activity outside that range. This can progress to an apparent reorganization of the cortical tonotopic map, which is reminiscent of the reorganization resulting from hearing loss restricted to a part of the hearing frequency range, although again, no hearing loss was apparent after our moderate-level sound exposure. Here, we review this work focusing specifically on the potential hearing problems that may arise despite a normally functioning auditory periphery.

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